High norepinephrine low dopamine reddit Nothing is interesting. Here's how to tell if you have a deficit. If SSRIs raise prolactin, then it is because the high serotonin levels are preventing the release of dopamine to combat prolactin levels, therefore the existence of elevated dopamine levels is not causing low dopamine, it is caused by low dopamine. Spectrum depression/anxiety. Music is usually the easiest way to feel it. Study on dopamine and anxiety. When you retain/abstain your body in response upregulates Androgen receptors (AR) and Dopamine (D2) receptors which in return makes you more sensitive/receptive to that hormone/and or neurotransmitter if their levels were to rise. Drugs like ecstasy or molly blow all your dopamine levels and take many days to replenish. It's actually a norepinephrine dopamine reuptake inhibitor. I think the idea will be that dopamine beta-hydroxylase catalyses the conversion of dopamine to norepinephrine, but if one boosts COMT activity with SAMe, more dopamine will go through COMT's degradation pathway instead (converting to 3-methoxytyramine then homovanillic acid) Thus, low levels of Norepinephrine are often associated with a specific PERCEPTUAL Disturbance, which may present differently depending on the Dopamine level. Role of Norepinephrine in Euphoria Produced by Stimulants (Norepinephrine Role in Euphoric Stimulants) The following is copy pasted from the Area 1255 blogspot, I can't post a direct link on Reddit for some reason. Apparently low levels of PEA can prevent dopamine from converting to norepinephrine. Below are excerpts from journal articles about VDR and dopamine signaling. I have hyperadrenergic orthostatic hypotension which is NOT the same as hyperPOTS (as I understood it, the difference being that my blood is hyperadrenergic all the time and only exacerbated but not actually caused by position changes). > Dopamine much more strongly than your 5HT subtypes. Reply reply Look up internasal bromantane, you’ll find info here on reddit. Something really interesting and related to learn about: The Yerkes-Dodson Law states that for a difficult task, if arousal (related to norepinephrine and dopamine concentrations) is too low, performance is weak because the task is perceived as too difficult, but if arousal is too high, the task is too stressful and performance suffers. such as eggs for this purpose, because they are versatile and appeal to some people As a result, the dopamine cannot be taken up so well. With time slowly i got back to exercice but not back to real training. It was a stomach bug. yes, for about 6-8 hours everyday after taking methylene blue, cyproheptadine, and eating well. Get the Reddit app Scan this QR code to download the app now It's reductionist and overly simplistic to reduce any disorder as being characterized by low or high levels of a neurotransmitter. Zinc will regulate copper levels. Not all types of anxiety/depression are the same. Also TRD for 30 years so that tracks. Some people with sleep problems are using low dose yohimbine - which elevates Norepinephrine, or noradrenaline, is essential to helping your brain send signals out to your body. It also reduces age-related loss of dopamine receptors (90). Zyban. It upregulates dopamine receptors and sensitizes them. which as it comes more in normal physiologic strength, helps dopamine in providing a higher level of libido, sex drive, and an emotional high. We also tend to have more dopamine in our brains than most people, leading to increased goal directed activity. It has a stronger effect on norepinephrine than dopamine, which is why it's not as effective for ADHD High levels of dopamine causes the feeling of euphoria. Like very intense boredom but an inability to do anything about it. norepinephrine, dopamine, acetylcholine at the same time. View community ranking In the Top 5% of largest communities on Reddit. Thank you for your detailed response! Yes, I've managed to get my serotonin and dopamine levels up through treatments, including taking tyrosine, but there still seems to be an issue in the conversion of dopamine into these adrenal hormones (although I'll admit, my dopamine is a little high now). If you are very low in iron you could become deficient in both. At high doses, quetiapine starts blocking significant amounts of dopamine receptors, norquietiapine is also a norepinephrine reuptake inhibigof (58 ki values) which can be stimulating for some" Anectodally, from my point of view doses below 200mg are barely touching D2 and if you add another 5-HT2A antagonism like Mirtazapine or Trazodone you Get the Reddit app Scan this QR code to download the app now. Promoting dopamine. A HIGH Dopamine but LOW Norepinephrine State typically I have trouble with high histamine already. Stimulants are largely said to work by increasing arousal (norepinephrine and dopamine) in the brain. Through overcompensation of the negatives of adenosine, you have dopamine and serotonin suppression causing them to sensitize, and through elevated levels of glutamate your mind counter balances by sensitizing GABA, and finally elevated norepinephrine once again overcompensates. Pheochromocytoma is a tumor that secretes epinephrine and norepinephrine, you diagnose it with a urine test looking for elevated levels of metabolites of these substances (VMA). Dopamine is released in conjunction with your body's movement towards acquiring something (food, sex, water, drug). Low levels of epinephrine can also contribute to weight gain and poor concentration. Try searching "dopamine serotonin homeostasis". Also regulates body movement and posture. It’s made by a researcher here on reddit, sirsadalot. Same can be done with dopamine (homovanillic acid). Lack of dopamine causes depression, low energy, lack of motivation. My guess is that the serotonin activity, besides the entactogen effect, intensifies the elevated mood from the dopamine effects (this is just speculation tho). You want cortisol low at night so you don’t take it then, you take it with breakfast and lunch. (Like peripheral necrosis. Thus, the binding of dopamine to these particular receptors inhibits the pre-synaptic release of dopamine. Yes, but it's not that your levels are low. When the poor dopamine can't get in, it will be back in the synaptic gap to connect to the opposite cell. With regards to libido, my very basic understanding is that high dopamine increases libido and high serotonin reduces it. I’ve been told no adaptogens for low cortisol! You want adrenal correct extract, and probably lots of it. It's when neurotransmitters are dumped into the synapse and stay there, that the receptors will down regulate so it takes more neurotransmitter to cause Eating certain foods, like those high in protein, and healthy practices like exercising and sleeping the recommended amount can help increase your body’s dopamine levels without medication. Most days i just feel flat and lack motivation. You need iron to make both tryptophan hydroxylase and tyrosine hydroxylase, required to synthesize serotonin and dopamine respectively. The average person has an extremely high tolerance for dopamine, meaning that they have to keep upping the stakes to get the feeling of satisfaction for a Dopamine hit. This drug has a strong risk, and the dosage should be as low as possible I decided to delve deeper into possible biological explanations behind the beneficial effects of long term semen retention and celibacy. This is particularly true for poeple overmethylating . Get the Reddit app Scan this QR code to download the app now Dopamine gives a 'high' and responsible for addiction to certain drugs. However, Wellbutrin increases dopamine and norepinephrine, and it made my OCD worse. Very high norepinephrine usually results from an underlying health problem. low dopamine, high norepinephrine etc, due to a mix of genetics and nurture and I wonder if low oxytocin can really damage people Get the Reddit app Scan this QR code to download the app now Secondly, the research on antidepressants found that the higher the affinity for dopamine transporters, the more likely it is to cause hyperhidrosis, which is a common side effect of many antidepressants. I think that having low MAOA, but normal MAOB activity, would lead mainly to increased norepinephrine and serotonin and less dopamine. Too little actual can resulr in addictive behaviors and depression. Bloodwork showed slightly elevated cortiso. I’m easily addicted and seek dopamine. Watch his video on dopamine and the analogy of the tidal wave pool. Or check it out in the app stores What causes of anhedonia, low dopamine or low norepinephrine, or others? Share Add a Comment. Glutathione levels. Because of this, your brain is making you frantically search for solutions to said deficiency, hence the hyperactivity, attention issues, and/or issues with executive function Basically, in addition to everything everybody else has posted, there's a fundamental difference between dopamine and serotonin. That along with too much cortisol. I'll copy it below. I've just read that high norepinephrine inhibits erection. Interestingly, the researchers found that these dopamine receptors only appear in the pineal gland towards the end of the night, as the dark period No desire to do anything. Learned this the hard way recently when my Dr's were clueless, knew I was taking 5-HTP morning and night and after 2 years I got really high blood pressure and bipolar like symptoms from serotonin eating up my dopamine and getting out of balance. As for dopamine, I do personally believe that activity that causes high dopamine spikes, such as porn, drugs, etc, can contribute to mental illness. The issue stems from having generally lower stimulus to the brain, and therefore seeking activities and substances that will provide it easily. Are chronic, abnormally low dopamine levels more associated with anxiety, or are chronic, abnormally high levels more associated with anxiety? Isn't there a hypothesis that patients with schizophrenia have highly elevated levels of I have tested high for dopamine in urine, suspect high for norepinephrine but not tested. We recommend browsing /r/adhd on desktop for the best experience. The issue is most likely, although you can keep a low flow going with distractions, the brain will continuously want something bigger, and deeper, stuff that comes from hard work and focus. My digestion wasn#t the best either. And when I take tryptophan (to raise serotonin), it works (I get very sociable/happy/good mood/flexible), but then my ability to focus seems to deteriorate (lowered dopamine, serotonin dominance). Really fatigued, couldn#t work out, no appetite. Most actions for being responsible are high-executive low-reward tasks, so they're very difficult to do There's a great episode on Dopamine on the Huberman Lab podcast where he talks about this. I'm not looking to get high lol just curious becuase I have low dopamine (I also have very low epinephrine) I’m also diagnosed with ADHD and anhedonia and that’s more common with low dopamine. Especially at 150mg, Wellbutrin is not acting on dopamine. High/low glutamate levels can cause issues. High/low serotonin, dopamine, norepinephrine, etc. Or check it out in the app stores - Norepinephrine will increase the afterload while increasing the Contractility. The low dopamine and norepinephrine levels really are a different but strong kind of pain . So, from a libido perspective, you'd want to maximize dopamine, but lower serotonin. Some need to reduce (antipsychotics, lamotrogine). Which shut down dopamine activity and give the peaceful state of mind. In the first few months of posts there, before the sub got popular Increasing dopamine without increasing norepinephrine is difficult because norepinephrine is synthesised from dopamine. There are so many things that can cause the variety of symptoms we have, and i've seen papers on LC affecting all of these areas. Low serotonin means low noradrenaline means fluctuating mood. One natural antidepressant is to increase dopamine by eating protein-rich foods. The only substances that affect all 3 in noticeable levels are dopaminergic stimulants. That serotonin (or at least the 5-HT_2 receptors) is more related to mood in this sense (sensitivity) would also explain why therapeutic use of things like MDMA and Psilocybin is Norepinephrine is a more local transmitter, but basically does the same stuff. Elevated levels of epinephrine can be factors contributing to restlessness, anxiety, sleep Researchers have found that intermittent fasting leads to higher levels of dopamine by increasing dopamine release and enhancing dopamine action (86-89). Yep im the same Reddit iOS Reddit Android Reddit Premium About Reddit Advertise Blog based on analogy with serotonin reuptake inhibitors, higher than 50% inhibition of DAT would be needed for the dopamine reuptake mechanism to be a significant mechanism of the drug's action. It can be confused with depression. Dopamine is also involved in attention, perception, motor function, and memory formation. (Alpha1, Beta1) then you are in a situation of low cardiac output with high SVR. •Stomach acid could be low, lending to SIBO and poor breakdown of aminos needed for neurotransmitter production •Poor absorption of nutrients from the SIBO needed to make dopamine •High oxidative stress (inflammation) impairing BH4 and dopamine creation •Strep (if present in high amounts in the SI) breaks amino acids into the wrong Get the Reddit app Scan this QR code to download the app now if you only triggered some of the dopamine and norepinephrine receptors but not enough of them due to low levels of dopamine and norepinephrine release than your body will not resupply as much of those And I'm 90lbs, five-foot-nothing - trying out high fat/low carb was a High dopamine is associated with psychosis, which is common in a mania, while low serotonin is associated with low mood. Therefore these meds are re-uptake inhibitors. . "Runner's high" is similar, it's actually quite mild and also really quite rare, if you get it it's usually with increases in Dopamine is the absolute prime way of lowering prolactin levels. [12] Not that the “dopamine counteracts the sexual numbing effect of SSRIs” has much empirical weight to begin with, but the number of doctors who prescribe 150mg of Wellbutrin to counteract sexual side effects via increasing dopamine levels is absurd. Vasopressin is pretty pointless on its own. Though you could try L-theanine it increases dopamine somewhat. Paired with coffee it gives a good boost in the morning Serotonin binds to this receptor, which inhibits these neurons from releasing dopamine and/or norepinephrine. I also have hypothyroidism anda hpa dysfunction, and both are known to increase norepinephrine. High amounts of dopamine are produced in the brain by drugs but then you’re more likely to get hooked leading you to try harder drugs with more dopamine and it’s a vicious cycle you’ll just keep chasing So if you have low adrenaline/noradrenaline levels or adrenal fatigue or high cortisol levels, you may experience low energy. Can anyone offer any assistance with how to help dopamine convert to norepinephrine? which some made a reddit sub about: r/hangovereffect. Interestingly, more recent research shows that serotonin is responsible for regulating noradrenaline, which itself is responsible for regulating mood. So if you have low dopamine it could result in high prolactin levels and also mess up the function of the pituitary gland which can trickle down to the adrenals. Rare tumors may uncontrollably produce catecholamines, and norepinephrine can rise during the process of drug withdrawal. Or check it out in the app stores TOPICS regular yoga practice has always been a go-to for naturally boosting my dopamine levels. As you seem to be aware, you are right, you're brain's anticipation of dopamine is quite high right now. High/low acetycholine levels. So dopamine or serotonin may temporarily help, but ultimately isn't the issue. The way you could criticize Reddit is that we weren't a company – we A drug that increases dopamine can reverse the effects of inflammation on the brain in depression This is actually interesting because using low dose stimulants like Adderall/ritalin is actually used to treat treatment resistent depression in geriatric populations (yes I am fully aware of the cardiovascular risks) the n is very, very The elevated norepinephrine signaling may then be accompanied by pro-inflammatory cytokine signaling as the brain becomes chronically elevated by stress signaling/norepinephrine. From every site I've seen on the symptoms of low serotonin and low dopamine, it's impossible to be low on both because the symptoms are complete opposites of each other. These are all controlled substances Edit: Forgot to get to your question about dopamine. This is an outdated mode of thinking about psychopathology. Or check it out in the app stores I may try low dose nasal spray but essentially don’t over do it and manage expectations. Hyperfocus may be because the dopamine is being made into norepinephrine (adrenaline Dopamine has a lot of involvement in reward and the expectation of reward, epinephrine and norepinephrine promote wakefulness and alertness and have some involvement in memory IIRC, etc. When you're hyperfixating or doing a low-executive high-reward task you have plenty of dopamine. With ADHD, have chronically low levels of certain chemicals (neurotransmitters like dopamine, norepinephrine and serotonin) because your brain is wired a bit differently. Hyperfocus: Elevated dopamine levels can lead to increased focus and concentration, often seen in conditions like ADHD where stimulant Well it says so on Wikipedia, with a good amount of literature references; "Sertraline (Zoloft) is a selective serotonin reuptake inhibitor (SSRI), but, uniquely among most antidepressants, it shows relatively high (nanomolar) affinity for the DAT as well. Too low dopamine signalling will cause hyperactivity due to restlessness (such as in ADHD), while too high dopamine signalling would cause hyperactivity due to novelty seeking / increased motivation (such as seen with recreational use of stimulants like cocaine). People with ADHD are thought to be deficient in primarily dopamine and norepinephrine. Some need more dopamine and more norepinephrine (SSRI/snri). In that case, you want to use dobutamine for the dial benefit of vasodilating Stimulants target norepinephrine. This is actually how it FEELS. Other neurotransmitters that seem off is very low PEA and high Tyrosine (without supplementation. I have high dopamine, high norepinephrine, low gaba. Then you have the D2 receptor, which acts as an autoreceptor (typically in one of its two isoforms) when expressed presynaptically. Drugs often cause higher levels of these neurotransmitters especially stimulants that it was a company that was built to be a company, and you could feel it in the product. could also achieve the same with the right dose of t3 and progesterone or any other pro metabolic substance. Since ADHD is characterized by low dopamine you have to put in work It does seem so. Caffeine and ephedrine are good for this, but their dopamine effect is not that great. Sort by: And with NE it can potentially be too high as well Reply reply Footsie_Galore Literally 30% of the times FNDs occur from simple things like borderline b12 and low b6, low zinc. You could also have sufficient Eggs: Research from the University of California, Berkeley suggests that people who suffer from depression have low amount of serotonin, norepinephrine and dopamine in their brains. e. Eating sugary sweet foods will cause a high spike in dopamine. One of the main areas of the brain where you'll find norepinephrine is a place called the locus ceruleus. CB1 also has heteromers with D2 dopamine receptors. Dopamine is very similar in structure to epi and norepi (they are all a category called catecholamines) but it's in the brain. There are also carcinoid tumors which secrete serotonin. Some people do better skipping directly to only adjusting norepinephrine (for example, they don’t do well with the side effects from increasing dopamine) or get a perfect balance by increasing dopamine or need to mix and match a bit with Get the Reddit app Scan this QR code to download the app now. That is how you start feeling those weird feelings. Is lower dopamine better then higher, is higher better then lower, or is too high/low bad and somewhere in the middle is what should be aimed for? Does higher/better levels of dopamine help study, completing assignments, decrease social anxiety, depression etc Are there ways to naturally increase dopamine? SAM-e can indeed raise both of those things (and yes, it can potentially raise norepinephrine as well, although you may not want to aim for extremely high norepinephrine, as that can contribute to anxiety). Prozac doesn’t interact with dopamine/its receptors directly, but the increased amount of serotonin in your brain fools the dopamine receptors in your brain into picking up the serotonin instead of dopamine, meaning there is more dopamine left in the junction between your neurons. Norepinephrine–dopamine reuptake inhibitor (NDRI) , major depressive disorder, atypical antidepressant , Epileptic seizures , ADHD, alleviate sexual dysfunction, weight loss, releasing agent of dopamine and norepinephrine (NDRA), similarly to other cathinones , Wellbutrin SR and XL According to my DUTCH test, my norepinephrine levels are on the higher end of the range (and my dopamine levels are quite low) . Especially since it's not being bound by dopamine and instead it's shared effect of CB1. But the main thing was super low nor-adrenalin and low adrenalin. Too much dopamine equals addictive behaviors and anxiety. For example: Wellbutrin is a stimulant which works on norepinephrine and dopamine. Dopamine reuptake inhibitors slow the refractory rate, allowing for longer attention spans As a person suffering from ADHD in my teens myself, you crave thrills and excitement as your brain has a low amount of dopamine in it. But I also don't know how dopamine would be low and norepinephrine would be high unless the enzyme DBH - which converts dopamine to norepi - had I know serotonin regulates dopamine levels; in a person w/ low serotonin (depressed) the addition of more dopamine alone will cause dopamine to be far too high which can cause symptoms of psychosis. So presumably the people who get dopamine agonists have unnaturally low dopamine (causing tremors) but acceptable serotonin levels to where this I think my body feels like low energy and then wants to get high calorie stuff in. I think D2 agonism would slowly cause upregulation of itself too. Adaptogens supposedly only help in the early stages of adrenal fatigue. ) high dopamine, very low DOPAC. There isn't any one solid answer but a few possibilities: The hyperactive component of ADHD can be due to low tonic (basal) dopamine levels, leaving the sufferer to (hyper)actively seek out reward (phasic dopamine release) by impulsively engaging in activities - when the tonic levels are raised (through reuptake inhibition by Ritalin) the "push" to seek this reward is decreased, Note: Not a biologist, just have spent quite a bit of time trying to figure things out. It can also cause you to experience anhedonia, or a lack of ability to feel pleasure, and cause Dopamine has a "bell-curve" (biphasic) effect on locomotion. In the later stages they can just lower your cortisol even more. Chronic kidney disease, in which high blood pressure is very common, also presents with high norepinephrine [1, 2, 3, 4]. Usually the mood elevating effects come from the fact that Niacin competes with other methyl takers and therefore high doses Niacin inhibits methylation mediated catecholamines => more dopamine serotonin norepinephrine available => better mood. I can answer from a more clinical perspective. Just Excess copper leads to higher epinephrine production, tanking dopamine. High Levels of Norepinephrine. Some meds work on dopamine, others on adrenaline or norepinephrine which is what they call it in the USA I believe, but the same thing. No "activation energy" to get up off the couch and start anything. BUT: When you first start taking zinc, it might make you feel like it's It controls sleep - deep sleep, if you have low levels of NE - you get higher cortisol - less sleep. My approech would be to calm/lower Norepinephrine (which balance dopamine on long term) and may increase Gaba with glutamine which seemed to work fine for me (better Just did a bunch of catecholamines serum testing and was a bit stunned to learn that my dopamine level is 2X the upper limit and I've got virtually no norepinephrine in my blood. Or check it out in the app stores but also exhibit a lot of the signs of low dopamine. I get so deficient in iron due to absorption issues that I get severe RLS from low dopamine. Doing low-effort things like sitting and scrolling fb/reddit, checking notifications, seeing if I got an upvote on something (mini dopamine). Going off the vyvanse without a substitute is an option, depending mostly of why you were prescribed it in the first place. Low levels are linked to depression, and high levels are linked to mania. Or check it out in the app stores Anxiety can be a byproduct of both too low and too high levels of dopamine & norepinephrine. Wellbutrin -Bupropion , all things related to this antidepressant. However, due to the increased exposure of hydroxybupropion over bupropion itself, which has higher affinity for the NET than the DAT,[105] bupropion's overall pharmacological profile in humans may end up making it effectively more of a norepinephrine reuptake inhibitor than a dopamine reuptake inhibitor. That's why people here take sometimes take cabergoline or other DA agonists to lower their prolactin levels. A benefit of the carnivore diet is that it eliminates high dopamine foods, and lowers your tolerance. [8][9][10] As such, it has been suggested that clinically it may weakly inhibit the reuptake of dopamine,[11] particularly at high dosages. You could also have high levels of these neurotransmitters causing your depression. Stop doing those first and your dopamine will come back and cortisol will lower. However, this seems like it would come at the expense of mental health. Below are some scientific findings: Dopamine: Increased Anxiety: High levels of dopamine can be linked to increased anxiety due to overstimulation of certain pathways in the brain. [29][30] Accordingly, the clinical We are on such high alert that our body sees freezing and dissociating as our only way to survive. You’re partially right in that SAM-e is relatively “new” as a supplement, yes — but it has always been naturally occurring in all human bodies in varying levels. Based on the journal articles below I'd say you're right about low dopamine levels due to the VDR taq (+/+). It basically sensitizes the receptors to the other pressors, so you can decrease the doses of the other pressors and limit the side effects from them. Get the Reddit app Scan this QR code to download the app now. But also psychotic effects. High functioning ADD folk (like myself) tend to be the ones who learned how to multi-process by shifting cognitive load among several pathways. It's kind of low, towards the back of the brain. A HIGH Dopamine but LOW Norepinephrine State typically presents with Changes in Mood; typically low Arousal and Depressed mood due to disorganized thought process and low blood pressure. Dopamine regulates prolactin (secreted by the pituitary gland). But the reason it feels that way is because of huge releases of dopamine AND norepinephrine, so you get body high euphoria through adrenal axis and dopamine influx. Boost dopamine and dopamine related functions and you indirectly improve norepinephrine levels as well. Second edit direct from the link: "When dopamine then interacts with its receptors, it inhibits the effects of norepinephrine—which means a decrease in the production and release of melatonin. This is what affects your mood, energy, alertness (norepinephrine) and feeling motivated to do things and everything you do feels great and fun I have very high epinephrine and norepinephrine levels and I really don’t want to take meds. But I don’t think exercise comes anywhere close to addictive behavior like that. In this clip we review reasons why abnormal dopamine levels can cause addiction in untreated individuals, and why psychostimulant treatment for ADHD and addiction disorders could be useful in offsetting the future addictive liability present in individuals with naturally low levels of dopamine in chief brain regions governing reward and Dopamine depends on dosing. I'll definitely check out the sub! Edit: I reread your post! If you want to avoid sexual side effects from serotoninergics, there are many other meds for mood and anxiety that work on cathecholeamines (nor and dopa). If I take too much ritalin (which boosts dopamine levels - I have ADHD), I get OCD, rigid, and inflexible (symptom of low serotonin, dopamine dominance). im moving to a higher altitude mountain town and the goal is to extend it to be a near permanent positive state. 1-5 dopamine (pointless), 5-10 beta, 10-15 alpha (backwards alphabet receptor, increasing dose). performance is maximized at a moderate amount of arousal. Norepinephrine is also highly linked to alertness and wakefulness. I have a lot of gene mutations for adhd also and the dopamine pathways. Norepinephrine --. The goal of medication is to increase the amount of these Although the affinity of D 2-like receptors for DA has been shown to be 10- to 100-fold greater than that of D 1-like receptors, at least in the striatum (Beaulieu and Gainetdinov, 2011), both high and low affinity states of D 1 and D 2 receptors exist and both exhibit similar nanomolar affinities for DA in their high affinity states [reviewed Get the Reddit app Scan this QR code to download the app now. Hi u/Afraid-Tumbleweed898 and thanks for posting on r/ADHD! The mobile apps used for Reddit are broken or are missing features that this subreddit depends on. It generally lifts mood. There's a theory called the Yerkes-Dodson law that says that performance can go down if arousal is either too low or too high, i. Then my symptoms startet to flare up. Does anyone know a herb or anything that can reduce these levels and that don’t interact with an ssri. kbgr srq lywhmb idycw jzqglkp moyps faij uttavz mmrr qbl yamqt rfuai sfsydues eihnwfoh rnl